Antibody-Mediated Resistance to Rhizomania Disease in Sugar Beet Hairy Roots
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چکیده
منابع مشابه
The hrpZ Gene of Pseudomonas syringae pv. phaseolicola Enhances Resistance to Rhizomania Disease in Transgenic Nicotiana benthamiana and Sugar Beet
To explore possible sources of transgenic resistance to the rhizomania-causing Beet necrotic yellow vein virus (BNYVV), Nicotiana benthamiana plants were constructed to express the harpin of Pseudomonas syringae pv. phaseolicola (HrpZ(Psph)). The HrpZ protein was expressed as an N-terminal fusion to the PR1 signal peptide (SP/HrpZ) to direct harpin accumulation to the plant apoplast. Transgene ...
متن کاملMolecular breeding for resistance to rhizomania in sugar beets
Lennefors, B-L. 2006. Molecular breeding for resistance to rhizomania in sugar beets. Doctor’s dissertation ISSN: 1652-6880, ISBN: 91-576-7255-5 Rhizomania is one of the most destructive sugar beet diseases. It is caused by Beet necrotic yellow vein virus (BNYVV) vectored by the soilborne protoctist Polymyxa betae Keskin. The studies in this thesis evaluated natural and transgenic resistances t...
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Rhizomania, caused by Beet necrotic yellow vein virus (BNYVV), severely impacts sugar beet (Beta vulgaris) production throughout the world, and is widely prevalent in most production regions. Initial efforts to characterize proteome changes focused primarily on identifying putative host factors that elicit resistant interactions with BNYVV, but as resistance breaking strains become more prevale...
متن کاملInhibition of water uptake in sugar beet roots by ammonia.
Ammonium sulfate, ammonium carbonate or ammonia gas inhibited water uptake in sugar beet roots whenever the pH was sufficiently high to cause the production of ammonia. When ammonia was removed by aeration, inhibition of the water uptake by roots was rapidly reversed. ATP at 0.2 mm appeared to either wholly or partially prevent the ammonia-induced inhibition of water uptake by roots. ATP may be...
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ژورنال
عنوان ژورنال: The Plant Pathology Journal
سال: 2019
ISSN: 1598-2254,2093-9280
DOI: 10.5423/ppj.oa.04.2018.0073